Lg Schematic Diagrams

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Lg Schematic Diagrams

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(A) In the absence of MCN1 activity (left), the dorsal gastric (DG) and lateral gastric (LG) neurons are silent, whereas Int1 produces bursting oscillations timed with the pyloric CPG (see Fig. 4.3). MCN1 stimulation (extracellular 1ms pulses at 15 Hz, amplitude at threshold) elicits bursting activity (period w10 s) in these neurons with DG and Int1 active out of phase with the LG neuron. (B) Schematic diagram shows that the axon terminals of MCN1 excite LG through a slow modulatory In the right panel, the solid arrow indicates the injected sensory neuron, and solid arrowheads indicate the coupled sensory neurons. The coupling site with the LG (open arrow) is proximal to the coupling site between the sensory neurons (open arrowhead).

N1,.N2, sensory nerves 1, 2 (modified from Herberholz et al. 2002, with permission from the Society for Neuroscience; see their fig. 3 for color images). (B) Schematic diagram of the LG escape circuit in the abdominal ganglia.In the study, a small quartz tube with about 0.1 to ~10 mg of lg was placed inside a larger quartz tube and pyrolyzed at 400 °C for 1 to 20 min. The products were collected from the wall The condensates collected from the tube wall contained low MW oxygenates, including 5hydroxymethylfurfural (5HMF), glycolaldehyde, hydroxylacetone, acetic acid and formic acid in addition to lg. The yields of low MW was also observed. The schematic diagram of the secondary reactions of lg is A, Schematic diagram of the gastric

mill.circuit. B. Stimulation of MCN l acti () C vates the gastric mill rhythm (period 7–15 s) in the crab. When MCN l is not active, LG is quiescent whereas Intl fires in pyloric time. in Ti | When MCN l is stimulated, LG and Intl os w Model cillate as a halfcenter oscillator. C, the (canonical) model was tuned to reproduce the LG 111111111111111111. ..". M.R.' known physiological behavior. (A & B *= adapted from Coleman et al.'). 5.5 McNisumulation Based on the result in step 2, draw the corresponding schematic diagram and then assign numbers to all links and joints, as shown in Figure 7.16(b). 4. Based on the result in step 3, transform the schematic diagram into the corresponding (5, 6) generalized kinematic

chain.as shown in Figure 7.16(c). (a) a b c d e f K Lc1 K Lc K Ld K Lg K Lf a b c d e f K Lc1 K Lc K Ld K Lg K Lf K Lc K Lc1 K Lg K Lf K Ld J G J R J R J R J R J G K Lc K Lc1 K Lg K Lf K Ld J G J R J R J R J R J G a b c d e f K These results suggest that a substantial amount of GABAergic tectothalamic neurons are found in all IC subdivisions and are the one of the output components of the IC's basic circuit. LG NEURONS WITH AXOSOMATIC ENDINGS ARE TECTOTHALAMIC CELLS GABAergic axons in the brachium of the IC have a larger diameter than nonGABAergic axons (Saint Marie et al., 1997). Semithin cross sections of the brachium of the IC immunostained for GABA revealed that GABAergic VI corticoLG cells: LG stimulation results in

disynaptic.activation of layer V neurons followed by trisynaptic EPSPs in layer VI neurons, and antidromic stimulation of terminal axons in the superior colliculus produces axonreflex EPSPs in layer VI corticoLG cells (Ferster and Lindström 1983). The IPSPs evoked in area B Unitary postsynaptic potentials demonstrated by spiketriggered averaging a Schematic diagram representing laminar location of source and target cells. Neuronal Topographic STM image of the 50 nm hFET together with the corresponding schematic diagram, (a) Topographic image taken at a sample bias voltage of 5 V. (b) Schematic diagram. Lg's of mFETs measured in this study were found to reflect the main feature of the Vth rolloff characteristic. Figure 2 shows

the.Vth rolloff characteristics of the nFET at Vd = 1.0 V and at Vd = 0.02 V. Note that the carrier profiles measured by STM are almost the same as the profiles when the electrical 95 Fig. 3.9: Simultaneous signal averaged intracellular recordings from LG and PDex show that GPR1 stimulation induces a slow, low amplitude EPSP in PD p. 97 Fig. 3.10: PD burst induction is not blocked by dtubocurarine. p. 100 Fig. 3.1 1 : GPRinduced PD/AB burst enhancement is not blocked by muscarinic antagonists. . p. 103 Fig. 3.12: Serotonin causes PD burst enhancement. p. 105 Fig. 4.1 : Schematic diagrams of the stomatogastric nervous system in the crab. p. 120 Fig.4.12 Accelerometer output Frequency spectrum with a 50Hz 50ug ac + lg dc input 93 4.13

Simulated.change in top and bottom air gap as the input dc level varies over lg to lg 94 5.1 Schematic diagram of the singleended switchedcapacitor sigma delta interface chip 98 5.2 Schematic diagram of the low noise singleended opamp 99 5.3 Die micrograph of the singleended switchedcapacitor sigmadelta modulator interface. The chip measures 2.0 mm x 1.2 mm in a standard 2P/1M 

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